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Alzheimer z disease advances in etiology, pathogenesis a by Khalid Iqbal, Sangram S. Sisodia, Bengt Winblad

By Khalid Iqbal, Sangram S. Sisodia, Bengt Winblad

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Extra resources for Alzheimer z disease advances in etiology, pathogenesis a therapeutics

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Neurology, 45, 957–62. Killiany RJ, Gomez-Isla T, Moss M, Kikinis R, Sandor T, Jolesz F, Tanzi R, Jones K, Hyman BT, Albert MS (2000) Use of structural magnetic resonance imaging to predict who will get Alzheimer’s disease. Ann Neurol, 47, 430–9. 10 EPIDEMIOLOGY AND RISK FACTORS Linn RT, Wolf PA, Bachman DL, Knoefel JE, Cobb JL, Belanger AJ, Kaplan EF, D’Agostino RB (1995) The ‘preclinical phase’ of probable Alzheimer’s disease: a 13-year prospective study of the Framingham cohort. Arch Neurol, 52, 485–90.

This was followed by the discovery of a polymorphic form of FE65 that is associated with increased susceptibility to late onset Alzheimer’s disease (LOAD). He has published evidence that two isoforms of FE65 are upregulated in relatively unaffected regions of the brain of patients with LOAD, and has emphasized the importance of such investigations as a means of detecting the earliest events in pathogenesis. Most of his other lines of research have also used genetics to further our understanding of the pathobiology of human aging.

Sisodia and Bengt Winblad Copyright & 2001 John Wiley & Sons Ltd Print ISBN 0-471-52176-0 Online ISBN 0-470-84645-3 1 The Transition from Normal Functioning to Dementia in the Aging Population LAURA FRATIGLIONI, BRENT SMALL, ¨ CKMAN BENGT WINBLAD AND LARS BA INTRODUCTION During the last few decades a substantial effort has been made to understand better the etiology and natural history of dementia, which has led to significant results. However, only in recent years has the availability of followup data from several community-based studies provided us with the opportunity to explore what happens just before the manifestation of the first dementia symptoms and before the diagnosis of dementia.

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